*I am not sharing the Jackie folder so no need to continue asking. Some are emails from people complaining about Ojai Raptor's care of Jackie. They included people's full names, addresses, phone numbers, email addresses. The Director has been very mean harassing anyone who dares question him or his treatment of Jackie. I don't want to give him a hit list of people to attack. He wasted countless hours sending nasty emails to people who didn't support him 100% while he was supposed to be caring for Jackie. They were really nasty. I didn't send in any emails. I posted my thoughts right here and on Facebook instead. He viciously attacked me right after I posted that I would request the full necropsy report if she died. That's all I posted at that time. I will redact the personal information of people who sent the emails then I'll post them.
If you take the files, please, indicate that you took them from here. Some people are saying they got the files from Ojai when they clearly did not. I added a mark to the files and can tell. They're also saying they got them from Fish & Wildlife from a list. They didn't come from them either at least not directly. I made this information act request within an hour of when Jackie died. Then I had to follow up multiple times to get the files. If you make a new one, you have to wait 90 days.
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Summary of the full necropsy report. Full six page report is linked below.
"Clinical decline and death were attributed to progressive debilitation from severe, chronic, necrotizing transmural ventriculitis complicated by secondary bacterial sepsis and terminal metabolic collapse. Chronicity of the ventriculitis was evidenced by extensive mural fibrosis and granulation tissue, likely
accounting for the bird's initial debilitation prior to presentation. Ventricular mucosal ulceration served as both the source of chronic hemorrhage and the portal of entry for secondary bacterial infection. Immunohistochemistry and in situ hybridization (ISH) demonstrated abundant intralesional influenza A virus antigen and RNA within multinucleated giant cells and infiltrating leukocytes, confirming a viral etiology for the ventriculitis. Furthermore, ISH revealed viral RNA within organizing thrombi in the heart and pectoral artery, and in a focus of necrotizing pneumonia and choroiditis of the left eye, indicating systemic viremic dissemination.
Bacterial sepsis secondary to the ulcerative ventriculitis was evidenced by vegetative endocarditis of the pulmonic valve and apical endocardium, fibrinonecrotizing arteritis and endophlebitis with thrombosis (right pectoral artery, caudal vena cava, hepatic vein), and intralesional bacilli in the caval
thrombus and bone marrow impression smears. Next-generation sequencing of the endocardial thrombus identified Escherichia coli and Clostridium perfringens as the predominant pathogens. Intralesional hair shafts, presumably from prey items, within the ventricular wall likely exacerbated and
perpetuated inflammation once the mucosa was ulcerated.
Refractory anemia while hospitalization likely resulted from ongoing hemorrhage from the ventricular ulcer compounded by anemia of chronic disease. Bone marrow cytology demonstrated erythroid maturational arrest, consistent with blunted erythropoietic compensation; the absence of viral RNA in the bone marrow by ISH argues against direct viral myelosuppression as the cause of the erythroid arrest. Progressive dehydration, reflected by renal urate stasis and renal gout, ultimately led to metabolic derangements that precipitated terminal decline.
Highly pathogenic avian influenza (HPAI) was confirmed by the California Animal Health and Food Safety Laboratory (CAHFS). Initial molecular confirmation and subtyping were complicated by low viral loads on choanal/cloacal swabs with high Ct values (35–38) on qPCR and negative virus isolation from tissues, consistent with resolving or chronic infection. However, subsequent qPCR using the pectoral artery thrombus confirmed HPAI H5/GsGd strain with lower Ct values (31–33) at CAHFS. Additionally, in-house targeted testing of the ventricular lesion for influenza A virus using formalin-fixed paraffin-embedded tissue revealed a high viral load with a low Ct value (25.6). The protracted disease course was likely facilitated by hospitalization and intensive supportive care.
HPAI manifesting predominantly as necrotizing ventriculitis is an atypical presentation in wild birds, with limited precedent in seabirds such as the great skua and European herring gull (Lean et al. 2023. Vet Pathol. 61(3):421–431). The absence of characteristic lesions in the central nervous system, pancreas, and spleen coupled with the lack of viral RNA in systemic lymphoid, endothelial, or epithelial tissues is likewise atypical. Furthermore, infection in early summer without concurrent local HPAI activity was unexpected; the nearest spatio-temporal detection occurred in Davis County, Utah, in mid-August.
A subacute, complete, displaced tracheal fracture with callus formation was also noted. While its cause remains undetermined, it likely stems from trauma that could have contributed to initial stranding and clinical compromise.
Toxicology screening revealed brodifacoum in the liver at 110 ppb, alongside background exposure to polychlorinated biphenyls (PCBs) and dichlorodiphenyldichloroethylene (DDE). Despite the chronic anemia and high hepatic anticoagulant rodenticide concentration, gross and histologic
Toxicology screening revealed brodifacoum in the liver at 110 ppb, alongside background exposure to polychlorinated biphenyls (PCBs) and dichlorodiphenyldichloroethylene (DDE). Despite the chronic anemia and high hepatic anticoagulant rodenticide concentration, gross and histologic
evidence of systemic thrombosis confirms the bird was not clinically coagulopathic; nonetheless, potential brodifacoum-induced immunomodulation cannot be ruled out as a factor in the severity of secondary sepsis. Enrofloxacin was detected in liver tissue via liquid chromatography–mass spectrometry, consistent with reported in-hospital antimicrobial therapy."
Full necropsy report. She wasn't euthanized. She died naturally on her own from dehydration as a result of the blood loss, infection, virus... She'd had avian flu in her system for a while. All the test results are in this report in this link.
I just added a new file with a visible watermark. The last watermark was invisible to most readers. The watermark does not interfere with capturing text. Please, cite where you got the file and data.
The purpose of getting the actual full necropsy is to find out what really happened. Ojai admitted they didn't know what was wrong with her or what to do for her. We can learn from the necropsy what was actually wrong with her. We can use this information to help bald eagles in the future. If you look at Ojai's report they only quarantined her the first two days. Should have been quarantined longer. They should have tested her for avian flu. Avian flu is in Big Bear lake where Jackie lived. Maybe she should have been given fluids for the dehyration. Maybe if they'd tested for avian flu immediately, they could have avoided the other stressful treatments and concentrated mainly on the virus and related symptoms.
Some people noticed that the report states they don't know if this is Jackie or not. They said that because Jackie wasn't tagged. This is most likely Jackie based on size, coloring, feathers per people who followed her closely for years. Plus, Jackie would not have left Shadow and her babies. Jackie was gone for two days per the cam. Then a female adult eagle is found down on the ground nearby. It's most likely Jackie. Jackie didn't fly away from her family. People on FB are saying a new eagle in the area is Jackie. People who know Jackie well states it's not.
CT scan result
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Full medical file from Ojai Raptor includes xrays
https://drive.google.com/file/d/1iOnq9-4U8fEnXBn2v-suwZK6V0ZFcdRk/view?usp=sharing
https://drive.google.com/file/d/1iOnq9-4U8fEnXBn2v-suwZK6V0ZFcdRk/view?usp=sharing
Radiology report
Bloods xl spread sheet
https://docs.google.com/spreadsheets/d/1jlw-MPGEeFfWbPtDfgU1bFGD9cJJvW5G/edit?gid=486812895#gid=486812895
https://docs.google.com/spreadsheets/d/1jlw-MPGEeFfWbPtDfgU1bFGD9cJJvW5G/edit?gid=486812895#gid=486812895
Hematology Cytology report. It's public now.
https://drive.google.com/drive/folders/1Bp1yZVrQ1o098J31rN9bbQ8upZoh8uLC
https://drive.google.com/drive/folders/1Bp1yZVrQ1o098J31rN9bbQ8upZoh8uLC
Ojai Raptor Center summary medical history
Someone sent an email as they were concerned whether or not Jackie was getting the best care. I have other emails where Ojai was contacting other raptor centers, LA Zoo asking for help and advice.
-----Original Message-----
Sent: Monday, August 10, 2026 8:34 AM
To: PermitsR8MB@fws.gov; Wildlife WHLAB <WILAB@wildlife.ca.gov>
Subject: Supplemental Urgent Request — Independent Veterinary Review and Potential Transfer of Bald Eagle Jackie
Dear Wildlife Health/Permit Official,
I am writing to supplement my earlier request concerning Jackie, the critically ill adult female bald eagle from Big Bear currently receiving treatment at Ojai Raptor Center.
I respectfully request that an agency veterinarian or other appropriately qualified independent veterinarian review Jackie's complete medical record as soon as possible and specifically determine whether her condition warrants immediate transfer to a tertiary veterinary hospital, university veterinary medical center, or other facility capable of providing a higher level of avian critical care.
Jackie's profound recurrent anemia has now persisted for approximately three weeks, and her packed cell volume has reportedly fallen below 6%. Despite extensive diagnostic evaluation, no underlying etiologic diagnosis has been publicly announced.
I recognize that the absence of a publicly disclosed diagnosis does not necessarily mean that her veterinary team lacks a working diagnosis or that disease-specific treatment is not occurring. This is precisely why I am requesting an independent review of the actual medical record rather than drawing conclusions from public updates.
I respectfully ask the reviewing agency to determine:
• Whether Jackie's current facility represents the appropriate level of care for the severity and complexity of her condition.
• Whether transfer to a tertiary or academic veterinary facility with advanced avian critical-care, hematology, infectious-disease, pathology, imaging and transfusion capabilities could provide diagnostic or therapeutic options not presently available.
• Whether her complete diagnostic and treatment chronology demonstrates appropriately timely escalation as her anemia persisted and repeatedly worsened.
• Whether any additional diagnostic testing, specialist consultation, transfusion strategy, disease-specific treatment or other intervention should be instituted immediately.
I am making this request while Jackie remains alive because any potentially beneficial intervention necessarily needs to occur before that opportunity is lost.
I am not asking the agency to presume negligence or wrongdoing. I am asking for independent professional oversight and an affirmative determination that every reasonable opportunity to diagnose, escalate and treat this federally protected bald eagle is being pursued.
Please document this request and forward it promptly to the veterinarian, wildlife-health official or permit authority with the ability to review Jackie's current care and determine whether additional intervention or transfer is warranted.
I would greatly appreciate written confirmation that this request has been received and reviewed.
Thank you for your attention to this urgent wildlife-welfare matter.
Respectfully,"
Doc file. This was already in the press release. "Postmortem examination summary for bald eagle 26-519
Mary Cummins of Animal Advocates is a wildlife rehabilitator licensed by the California Department of Fish and Game and the USDA. Mary Cummins is also a licensed real estate appraiser in Los Angeles, California.
The decline and death of this eagle were due to health complications from a chronic highly pathogenic avian influenza (HPAI) virus infection. The HPAI in this case likely caused an ulcer in the gizzard, which then resulted in anemia from ongoing bleeding and serving as an entry point for bacteria that spread to other organs, ultimately leading to dehydration and terminal metabolic changes. Additional laboratory testing revealed evidence of viral spread beyond the gizzard to other tissues. This presentation of HPAI - predominantly as a gizzard ulcer rather than the typical brain or multi-organ disease - is unusual in wild birds but has been reported in other species.
Routine toxicology screening revealed an anticoagulant rodenticide, brodifacoum, in the liver at 110 parts per billion, alongside low-level exposure to environmental contaminants (PCBs and DDE). Despite the anemia and elevated rodenticide concentration, the presence of multiple blood clots throughout the body confirms that the bird's clotting ability was likely not impaired. Additional testing included veterinary clinical pathologist interpretation of bone marrow and body fluid samples, genetic sequencing of a blood clot in the heart (which identified E. coli and C. perfringens as the primary bacterial infections), testing for West Nile virus (negative), heavy metal screening including lead (no significant findings), and liver vitamin E levels (normal).
"
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